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A grammar of Gyeli
This grammar offers a grammatical description of the Ngòló variety of Gyeli, an endangered Bantu (A80) language spoken by 4,000-5,000 "Pygmy" hunter-gatherers in southern Cameroon. It represents one of the most comprehensive descriptions of a northwestern Bantu language. The grammatical description, which is couched in a form-to-function approach, covers all levels of language, ranging from Gyeli phonology to its information structure and complex clauses. It draws on nineteen months of fieldwork carried out as part of the "Bagyeli/Bakola" DoBeS (Documentation of Endangered Languages) project between 2010 and 2014. The resulting multimodal corpus from that project, which includes texts of...
Confluence Narratives
Confluence Narratives: Ethnicity, History and Nation-Making in the Americas explores how a collection of contemporary novels calls attention to the impact of ethnicity on national identities in the Americas. These historical narratives portray the cultural encounters—the conflicts and alliances, peaceful borrowings and violent seizures—that have characterized the history of the American continents since the colonial period. In the second half of the twentieth century, North and South American readers have witnessed a steady output of novels that revisit moments of cultural confluence as a means of revising national histories. Confluence Narratives proposes that these historical novels, p...
Pediatric Neurology
Herpes simplex encephalitis (HSE) is the most common sporadic viral encephalitis in the Western world. The pathogenesis of HSE, which affects a small minority of HSV-1-infected individuals, has long remained elusive. Mendelian defects in the TLR3-interferon (IFN) and IFN-responsive pathways were recently shown to predispose to HSE, at least in some children. Autosomal recessive STAT-1 deficiency and X-linked NEMO deficiency were found in children with both mycobacterial disease and HSE. Autosomal recessive UNC-93B deficiency and autosomal dominant TLR3 deficiency were then described in children with isolated HSE. These discoveries provided proof-of-principle that HSE may result from a novel group of single-gene inborn errors of interferon (IFN)-mediated immunity. The TLR3–UNC-93B-dependent production of IFN-α/β and IFN-λ is essential to confer protective immunity to HSV-1 in the central nervous system during the course of primary infection in childhood.
Cancer Research
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