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This book covers state-of-the art techniques for high-level modeling and validation of complex hardware/software systems, including those with multicore architectures. Readers will learn to avoid time-consuming and error-prone validation from the comprehensive coverage of system-level validation, including high-level modeling of designs and faults, automated generation of directed tests, and efficient validation methodology using directed tests and assertions. The methodologies described in this book will help designers to improve the quality of their validation, performing as much validation as possible in the early stages of the design, while reducing the overall validation effort and cost.
This book covers state-of-the art techniques for high-level modeling and validation of complex hardware/software systems, including those with multicore architectures. Readers will learn to avoid time-consuming and error-prone validation from the comprehensive coverage of system-level validation, including high-level modeling of designs and faults, automated generation of directed tests, and efficient validation methodology using directed tests and assertions. The methodologies described in this book will help designers to improve the quality of their validation, performing as much validation as possible in the early stages of the design, while reducing the overall validation effort and cost.
This book constitutes the second part of the refereed proceedings of the Third International Conference, IC3 2010, held in Noida, India, in August 2010. The 23 revised full papers presented were carefully reviewed and selected from numerous submissions.
This book discusses the latest findings on the pathogenesis and treatment of IgA nephropathy. It particularly focuses on recently recognized initiation and progression factors and the varying treatment strategies in different regions, such as Asia, Europe, and the United States. More than 40 years have passed since Dr. Jean Berger first described primary IgA nephropathy (“Nephropathy with mesangial IgA-IgG deposits”) as a new disease entity. Immunohistopathologically, IgA nephropathy is characterized by the granular deposition of IgA (IgA1) and C3 in the glomerular mesangial areas with mesangial cell proliferation and the expansion of mesangial matrices. It is clear that IgA nephropathy ...