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Where Gallantry is Tradition
Reminiscences by alumni of the college.
The Bile Acids: Chemistry, Physiology, and Metabolism
Over a decade has elapsed since the last volume in this series was published. At that time we considered that we had comprehensively covered all aspects relating to bile acid chemistry and physiology. However, major strides have been made in our understanding of the physiology and pathophysiology of bile acids, due largely to the great advances which have taken place in analytical technology. As a result, the need to document these advances was felt acutely, and therefore this volume is devoted to methodologies in bile acid analysis and their applications. This volume includes twelve chapters written by prominent scientists in the field of bile acid research. The initial chapter discusses te...
Hypolipidemic Agents
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Progress in Medicinal Chemistry
Progress in Medicinal Chemistry
The Bile Acids, Chemistry, Physiology, and Metabolism
1 Mechanisms of Bile Acid Biosynthesis.- I. Introduction.- II. Formation of Cholic Acid.- A. Changes in Steroid Nucleus.- B. Oxidation of Side Chain.- III. Formation of Chenodeoxycholic Acid.- IV. Formation of Other Primary Bile Acids.- V. Conjugation of Bile Acids.- VI. Regulation of Bile Acid Formation.- VII. Formation of Bile Salts in "Primitive" Animals.- A. Changes in Steroid Nucleus.- B. Oxidation of Side Chain.- References.- 2 Bile Salt Transport Systems.- I. Introduction.- II. Active Transport in the Intestine.- III. Passive Proximal Intestinal Absorption of Bile Salts.- IV. Passive Ab.
Atherosclerosis IV
The presence of monotypism in thick atherosclerotic lesions of black females with G-6-PD mosaicism first reported by the Benditts (1973) has been confirmed in two other laboratories. However, we believe that it is premature to conclude that the finding of monotypism necessarily indicates monoclonal origin of athero sclerotic lesions. We have suggested two alternative explanations for the obser vation of monotypism which we believe must be shown to be invalid before accept ing monoclonal origin as the only plausible way to account for the observed G-6-PD monotypism. One of these two alternatives relates to clonal heterogeneity of cell growth potential, i. e. , during the course of progressive...
Annual Reports in Medicinal Chemistry
Annual Reports in Medicinal Chemistry
