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Emerging Concepts in Bacterial Biofilms
The ability to form biofilms is a universal attribute of bacteria. Bacteria are able to grow on almost every surface, forming these architecturally complex communities. In biofilms, the cells grow in multicellular aggregates, encased in an extracellular matrix produced by the bacteria themselves. They impact humans in many ways, and can form in natural, medical and industrial settings. For example, the formation of biofilms on medical devices such as catheters or implants often results in difficult-to-treat chronic infections. This book focuses on emerging concepts in bacterial biofilm research, such as the different mechanisms of biofilm formation in Gram negative and Gram positive bacteria, and the burden of biofilm associated infections. It also highlights the various anti-biofilm strategies that can be translated to curb biofilm-associated infections and the escalation of antimicrobial resistance determinants.
Microbial Linear Plasmids
A book that constitutes the first attempt to comprehensively assemble current knowledge of different types of such elements, highlight recent developments in the field, and challenge the distinction between viruses and linear plasmids. Linear plasmids of microbes represent a heterogenous group of extrachromosomal genetic elements initially assumed to be rare and peculiar. However, we now know that they are fairly frequently occurring plasmids in bacterial and eukaryotic species. Viral strategies to avoid shortening of the linear molecules during replication imply a common ancestry.
Gene der Chinaldin-Degradation in Arthrobacter ilicis Rü61a
!104411406! Chinaldin kann von Arthrobacter ilicis Rü61a als Kohlenstoff- und Energiequelle genutzt werden. Die Gene für die Chinaldin-4-Oxidase (Qox), eine Molybdopterin-Cytosin-Dinukleotid enthaltende molybdänhaltige Hydroxylase, sowie für die 1H-4-Oxochinaldin-3-Monooxygenase, die 1H-3-Hydroxy-4-Oxochinaldin-2,4-Dioxygenase und die N-Acetylanthranilat Amidase wurden im Genom von A. ilicis identifiziert. Die qox-Gene wurden erfolgreich in Pseudomonas putida KT2440 pKP1 heterolog expremiert. Der Austausch eines in den molybdänhaltigen Hydroxylasen strikt konservierten Gluamatratrestes gegen einen Glutaminrest führte zum nahezu vollständigen Verlust von Qox-Aktivität, was die Hypothe...
